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Research output

Inverse remodelling of K(2P)3.1 K(+) channel expression and action potential duration in left ventricular dysfunction and atrial fibrillation: implications for patient-specific antiarrhythmic drug therapy

Research output: Contribution to journal › Article › Academic › peer-review

Associated researcher

  • Schmidt, C.
  • Wiedmann, F.
  • Zhou, X.
  • Voigt, N.
  • Ratte, A.
  • Lang, S.
  • Kallenberger, S. M.
  • Campana, C.
  • Weymann, A.
  • De Simone, R.
  • Szabo, G.
  • Ruhparwar, A.
  • Kallenbach, K.
  • Karck, M.
  • Ehrlich, J. R.
  • Baczko, I.
  • Borggrefe, M.
  • Ravens, U.
  • Dobrev, D.
  • Katus, H. A.
  • Thomas, D.

Associated organisations

    Research areas

  • Arrhythmia, Atrial fibrillation, Electrical remodelling, Electrophysiology, Heart failure, K(2P)3.1 channel, HEART-FAILURE, UP-REGULATION, MODEL, SUPPRESSION, TACHYCARDIA, MECHANISMS

  1. Related by authors

  2. Computational modeling: What does it tell us about atrial fibrillation therapy?

    Research output: Contribution to journal › Review article › Academic › peer-review

  3. Postoperative atrial fibrillation: mechanisms, manifestations and management

    Research output: Contribution to journal › Review article › Academic › peer-review

  4. German Cardiac Society Working Group on Cellular Electrophysiology state-of-the-art paper: impact of molecular mechanisms on clinical arrhythmia management

    Research output: Contribution to journal › Review article › Academic › peer-review

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Details

Original languageEnglish
Pages (from-to)1764-1774
Number of pages11
JournalEuropean Heart Journal
Volume38
Issue number22
DOIs
Publication statusPublished - 7 Jun 2017

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