INIS
ganglions
100%
phenotype
100%
gain
100%
mice
100%
roots
100%
pain
100%
neurons
100%
fibers
83%
patients
50%
losses
33%
control
16%
comparative evaluations
16%
nerves
16%
mutations
16%
sodium
16%
modeling
16%
in vivo
16%
Biochemistry, Genetics and Molecular Biology
Wild Type
100%
Gain of Function Mutation
100%
Sodium Channel
100%
Sensory Neuron
100%
Channelopathy
100%
Multielectrode Array
100%
Whole Cell Patch Clamp
100%
Mechanical Hyperalgesia
100%
Thermal Hyperalgesia
100%
Keyphrases
Nav1.7
100%
Dorsal Root Ganglion Neurons
100%
Hyperexcitability
100%
Pain Phenotype
100%
Gain-of-function Variant
100%
Knock-in Mouse Model
42%
Small Fiber Neuropathy
42%
Fiber Loss
28%
Gain-of-function mutation
14%
Neuropathy
14%
Littermate Controls
14%
Neuropathic Pain
14%
Intraepidermal Nerve Fiber Density
14%
Sodium Channel
14%
Hyperexcitable
14%
Unmyelinated
14%
Channelopathy
14%
Sensory Dysfunction
14%
Pain Disorder
14%
Thermal Hyperalgesia
14%
Sensory Neurons
14%
Myelinated Fibers
14%
Mechanical Hyperalgesia
14%
Multi-electrode Array Recordings
14%
Whole-cell Patch-clamp
14%
Medicine and Dentistry
Dorsal Root Ganglion
100%
Small Fiber Neuropathy
75%
Psychogenic Pain
25%
Neuropathic Pain
25%
Gain of Function Mutation
25%
Sodium Channel
25%
Channelopathy
25%
Sensory Dysfunction
25%
Neuropathy
25%
Nerve Fiber
25%
Patch Clamp
25%
Whole Cell
25%
Multielectrode Array
25%
Thermal Hyperalgesia
25%
Mechanical Hyperalgesia
25%
Neuroscience
Dorsal Root Ganglion
100%
Neuropathy
100%
In Vivo
25%
Nerve Fiber
25%
Hyperalgesia
25%
Sodium Channel
25%
Neuropathic Pain
25%
Channelopathy
25%
Patch Clamp
25%
Psychogenic Pain
25%
Multielectrode Array
25%
Gain of Function Mutation
25%
Sensory Neuron
25%
Immunology and Microbiology
Ganglion
100%
Dorsal Root
100%
Wild Type
25%
Sodium Channel
25%
Gain of Function Mutation
25%
Nerve Fiber
25%
Whole Cell Patch Clamp
25%
Mechanical Hyperalgesia
25%
Thermal Hyperalgesia
25%