Thirty-Eight-Negative Kinase 1 Is a Mediator of Acute Kidney Injury in Experimental and Clinical Traumatic Hemorrhagic Shock

Rebecca Halbgebauer, Ebru Karasu, Christian K Braun, Annette Palmer, Sonja Braumüller, Anke Schultze, Fabian Schäfer, Sarah Bückle, Alica Eigner, Ulrich Wachter, Peter Radermacher, Ranillo R G Resuello, Joel V Tuplano, Kristina Nilsson Ekdahl, Bo Nilsson, Milena Armacki, Alexander Kleger, Thomas Seufferlein, Miriam Kalbitz, Florian GebhardJohn D Lambris, Martijn van Griensven, Markus Huber-Lang

Research output: Contribution to journalArticleAcademicpeer-review

Original languageEnglish
Article number2081
Number of pages12
JournalFrontiers in Immunology
Volume11
DOIs
Publication statusPublished - 26 Aug 2020

Keywords

  • trauma
  • injury
  • TNK1
  • AKI
  • complement
  • inflammation
  • IL-6
  • blood loss
  • COMPLEMENT ACTIVATION
  • INHIBITION
  • C3

Cite this

Halbgebauer, R., Karasu, E., Braun, C. K., Palmer, A., Braumüller, S., Schultze, A., Schäfer, F., Bückle, S., Eigner, A., Wachter, U., Radermacher, P., Resuello, R. R. G., Tuplano, J. V., Nilsson Ekdahl, K., Nilsson, B., Armacki, M., Kleger, A., Seufferlein, T., Kalbitz, M., ... Huber-Lang, M. (2020). Thirty-Eight-Negative Kinase 1 Is a Mediator of Acute Kidney Injury in Experimental and Clinical Traumatic Hemorrhagic Shock. Frontiers in Immunology, 11, [2081]. https://doi.org/10.3389/fimmu.2020.02081