The majority of the genetic risk for Paget's disease of bone is explained by genetic variants close to the CSF1, OPTN, TM7SF4, and TNFRSF11A genes

Pui Yan Jenny Chung, Greet Beyens, Steven Boonen, Socrates Papapoulos, Piet Geusens, Marcel Karperien, Filip Vanhoenacker, Leon Verbruggen, Erik Fransen, Jan Van Offel, Stefan Goemaere, Hans-Georg Zmierczak, Rene Westhovens, Jean-Pierre Devogelaer, Wim Van Hul

Research output: Contribution to journalArticleAcademicpeer-review

Original languageEnglish
Pages (from-to)615-626
JournalHuman Genetics
Volume128
Issue number6
DOIs
Publication statusPublished - Dec 2010

Cite this

Chung, P. Y. J., Beyens, G., Boonen, S., Papapoulos, S., Geusens, P., Karperien, M., Vanhoenacker, F., Verbruggen, L., Fransen, E., Van Offel, J., Goemaere, S., Zmierczak, H-G., Westhovens, R., Devogelaer, J-P., & Van Hul, W. (2010). The majority of the genetic risk for Paget's disease of bone is explained by genetic variants close to the CSF1, OPTN, TM7SF4, and TNFRSF11A genes. Human Genetics, 128(6), 615-626. https://doi.org/10.1007/s00439-010-0888-2