The deletion mutant EGFRvIII significantly contributes to stress resistance typical for the tumour microenvironment

J. Theys*, B. Jutten, L.J. Dubois, K.M.A. Rouschop, R.K. Chiu, Y. Li, K. Paesmans, P. Lambin, G. Lammering, B.G. Wouters

*Corresponding author for this work

    Research output: Contribution to journalArticleAcademicpeer-review

    Original languageEnglish
    Pages (from-to)399-404
    Number of pages6
    JournalRadiotherapy and Oncology
    Volume92
    Issue number3
    DOIs
    Publication statusPublished - Sep 2009

    Keywords

    • Epidermal growth factor receptor (EGFR)
    • EGFRvIII
    • Tumour
    • Microenvironment
    • Hypoxia
    • GROWTH-FACTOR RECEPTOR
    • GLIOBLASTOMA CELLS
    • RADIOTHERAPY
    • INHIBITION
    • CANCER
    • PATHWAY
    • XENOGRAFTS
    • ACTIVATION
    • EXPRESSION
    • CETUXIMAB

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