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Telomere shortening in chronic obstructive pulmonary disease.
J.M. Houben
*
, E.M. Mercken
, H.B. Ketelslegers
, A. Bast
,
E.F. Wouters
,
G.J. Hageman
,
A.M. Schols
*
Corresponding author for this work
CAPHRI - Care and Public Health Research Institute
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:
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Keyphrases
Chronic Obstructive Pulmonary Disease
100%
Telomere Length
100%
Telomere Shortening
100%
Chronic Obstructive Pulmonary Disease Patient
80%
Oxidative Stress
40%
Negatively Associated
40%
Superoxide Dismutase
40%
Genotype
20%
Age-matched
20%
Age-adjusted
20%
Chronic Disease
20%
Systemic Inflammation
20%
Inflammatory Markers
20%
Lung Function
20%
Disease Severity
20%
Moderate to Severe
20%
Severe Chronic Obstructive Pulmonary Disease
20%
Pulmonary Infection
20%
Fat Mass
20%
Pulmonary Disease
20%
Smoking History
20%
Antioxidant Enzymes
20%
Fat Mass Percentage
20%
Superoxide Dismutase Activity
20%
Oxidant-antioxidant Balance
20%
Chronic Oxidative Stress
20%
Oxidative Inflammation
20%
INIS
diseases
100%
telomeres
100%
patients
66%
length
55%
control
33%
inflammation
33%
oxidation
33%
superoxide dismutase
33%
fats
22%
antioxidants
22%
data
11%
levels
11%
lungs
11%
receptors
11%
comparative evaluations
11%
pathology
11%
genotype
11%
enzymes
11%
balances
11%
age groups
11%
oxidants
11%
preservation
11%
Neuroscience
Chronic Obstructive Pulmonary Disease
100%
Telomere Shortening
100%
Telomere
71%
Oxidative Stress
42%
Superoxide Dismutase
42%
Antioxidant
28%
Oxidizing Agent
14%
Receptor
14%
Biochemistry, Genetics and Molecular Biology
Telomere Shortening
100%
Telomere Length
100%
Oxidative Stress
60%
Superoxide Dismutase
60%
Fat Mass
40%
Genotyping
20%
Antioxidant Enzyme
20%
Immunology and Microbiology
Telomere Length
100%
Telomere Shortening
100%
Oxidative Stress
60%
Superoxide Dismutase
60%
Fat Mass
40%
Systemic Inflammation
20%
Pharmacology, Toxicology and Pharmaceutical Science
Chronic Obstructive Lung Disease
100%
Superoxide Dismutase
42%
Inflammation
28%
Antioxidant
28%
Receptor
14%
Oxidizing Agent
14%
Disease Severity
14%
Chronic Disease
14%