Spatial lipidomics reveals biased phospholipid remodeling in acute Pseudomonas lung infection

Alison J. Scott*, Shane R. Ellis, Casey E. Hofstaedter, Ron M.A. Heeren, Robert K. Ernst*

*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

Abstract

Pseudomonas aeruginosa (Pa) is a pathogen causing chronic pulmonary infections in patients with cystic fibrosis (CF). Manipulation of lipids is an important feature of Pa infection and on a tissue-level scale is poorly understood. Using a mouse model of acute Pa pulmonary infection, we explored the whole-lung phospholipid response using mass spectrometry imaging (MSI) and spatial lipidomics. Using a histology-driven analysis, we isolated airways and parenchyma from both mock- and Pa-infected lungs and used systems biology tools to identify enriched metabolic pathways from the differential phospholipid identities. Infection was associated with a set of 26 ions, with 11 unique to parenchyma and 6 unique to airways. Acyl remodeling was differentially enriched in infected parenchyma as the predominant biological function. These functions correlated with markers of polymorphonuclear (PMN) cell influx, a defining feature of the lung response to Pa infection, implicating enzymes active in phospholipid remodeling.
Original languageEnglish
Article number107700
Number of pages11
JournaliScience
Volume26
Issue number9
DOIs
Publication statusPublished - 15 Sept 2023

Keywords

  • Biological sciences
  • Lipidomics
  • Microbiology

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