SLC44A2 deficient mice have a reduced response in stenosis but not in hypercoagulability driven venous thrombosis

Julia Tilburg, Danielle M. Coenen, Gaia Zirka, Sophie Dolleman, Annemarie M. van Oeveren-Rietdijk, Mieke F. A. Karel, Hetty C. de Boer, Judith M. E. M. Cosemans, Henri H. Versteeg, Pierre E. Morange, Bart J. M. van Vlijmen*, Chrissta X. Maracle, Grace M. Thomas

*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

Original languageEnglish
Pages (from-to)1714-1727
Number of pages14
JournalJournal of Thrombosis and Haemostasis
Volume18
Issue number7
DOIs
Publication statusPublished - Jul 2020

Keywords

  • animal models of human disease
  • basic science research
  • thrombosis
  • vascular biology
  • vascular diseases
  • VON-WILLEBRAND-FACTOR
  • DEEP-VEIN THROMBOSIS
  • TRANSPORTER-LIKE PROTEIN-2
  • FACTOR-BINDING
  • NEUTROPHILS
  • GLYCOPROTEIN
  • INDIVIDUALS
  • INITIATE
  • LOCI
  • LUNG

Cite this

Tilburg, J., Coenen, D. M., Zirka, G., Dolleman, S., van Oeveren-Rietdijk, A. M., Karel, M. F. A., de Boer, H. C., Cosemans, J. M. E. M., Versteeg, H. H., Morange, P. E., van Vlijmen, B. J. M., Maracle, C. X., & Thomas, G. M. (2020). SLC44A2 deficient mice have a reduced response in stenosis but not in hypercoagulability driven venous thrombosis. Journal of Thrombosis and Haemostasis, 18(7), 1714-1727. https://doi.org/10.1111/jth.14835