Review article: hyperammonaemic and catabolic consequences of upper gastrointestinal bleeding in cirrhosis.

INIS

• reviews 100%
• proteins 100%
• ammonia 100%
• patients 66%
• therapy 66%
• kidneys 66%
• excretion 66%
• infusion 66%
• liver 33%
• values 33%
• simulation 33%
• amino acids 33%
• metabolism 33%
• production 33%
• failures 33%
• synthesis 33%
• stimuli 33%
• precursor 33%
• molecules 33%
• hemoglobin 33%
• leucine 33%
• half-life 33%
• precipitation 33%
• catabolism 33%
• valine 33%
• ornithine 33%

Keyphrases

• Cirrhosis 100%
• Catabolic 100%
• Upper Gastrointestinal Bleeding 100%
• Leucine 80%
• Bleeding 80%
• Hyperammonemia 60%
• Ammoniagenesis 60%
• Clinical Treatment 20%
• Large Intestine 20%
• Amino Acids 20%
• Hemoglobin 20%
• Small Intestine 20%
• Therapeutic Implications 20%
• Intravenous Infusion 20%
• Liver Cirrhosis 20%
• Pharmacological Therapy 20%
• Life-threatening Complications 20%
• Catabolism 20%
• Valine 20%
• Protein Synthesis 20%
• Dividing Cells 20%
• Clotting Factors 20%
• Ornithine 20%
• Short Half-life 20%
• Ammonia Production 20%
• Therapeutic Failure 20%
• Ammonia Excretion 20%
• Ammonia Metabolism 20%
• Inter-organ 20%

Medicine and Dentistry

• Liver Cirrhosis 100%
• Upper Gastrointestinal Bleeding 100%
• Ammonium 60%
• Ciclonicate 60%
• Isoleucine 60%
• Hyperammonemia 60%
• Precursor 20%
• Infusion 20%
• Intravenous Drug Administration 20%
• Drug Therapy 20%
• Leucine 20%
• Gut 20%
• Protein Synthesis 20%
• Biological Product 20%
• Bleeding 20%
• Blood Clotting Factor 20%
• Ornithine 20%
• Essential Amino Acid 20%
• Valine 20%
• Large Intestine 20%