Protective role of chaperone-mediated autophagy against atherosclerosis

Julio Madrigal-Matute, Jenny de Bruijn, Kim van Kuijk, Dario F Riascos-Bernal, Antonio Diaz, Inmaculada Tasset, Adrián Martín-Segura, Marion J J Gijbels, Bianca Sander, Susmita Kaushik, Erik A L Biessen, Simoni Tiano, Mathieu Bourdenx, Gregory J Krause, Ian McCracken, Andrew H Baker, Han Jin, Nicholas E S Sibinga, Jose Javier Bravo-Cordero, Fernando MacianRajat Singh, Patrick C N Rensen, Jimmy F P Berbée, Gerard Pasterkamp, Judith C Sluimer*, Ana Maria Cuervo*

*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

Abstract

Significance Cardiovascular diseases remain the leading cause of death worldwide, with atherosclerosis being the most common source of clinical events. Metabolic changes with aging associate with concurrent increased risk of both type 2 diabetes and cardiovascular disease, with the former further raising the risk of the latter. The activity of a selective type of autophagy, chaperone-mediated autophagy (CMA), decreases with age or upon dietary excesses. Here we study whether reduced CMA activity increases risk of atherosclerosis in mouse models. We have identified that CMA is up-regulated early in response to proatherogenic challenges and demonstrate that reduced systemic CMA aggravates vascular pathology in these conditions. We also provide proof-of-concept support that CMA up-regulation is an effective intervention to reduce atherosclerosis severity and progression.

Original languageEnglish
Article numbere2121133119
Number of pages12
JournalProceedings of the National Academy of Sciences of the United States of America
Volume119
Issue number14
DOIs
Publication statusPublished - 5 Apr 2022

Keywords

  • CHOLESTEROL
  • DEATH
  • DEFICIENCY
  • DEGRADATION
  • INSULIN-RESISTANCE
  • MACROPHAGES
  • PLAQUES
  • PROTEIN
  • RECEPTOR
  • SMOOTH-MUSCLE-CELLS
  • atherosclerotic plaques
  • lipid challenge
  • lysosomes
  • proteolysis
  • vascular disease

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