INIS
regulations
100%
muscles
100%
mice
100%
aging
100%
phosphodiesterases
100%
levels
80%
humans
80%
instability
60%
lungs
40%
nucleotides
40%
inhibition
40%
sodium
40%
blood pressure
40%
vascular diseases
40%
cyclases
40%
comparative evaluations
20%
genes
20%
losses
20%
populations
20%
messenger-rna
20%
cell cultures
20%
proteins
20%
genetics
20%
thickness
20%
kinases
20%
hypothesis
20%
dbp
20%
rings
20%
aorta
20%
hypertrophy
20%
nitric oxide
20%
excision repair
20%
hydrolysis
20%
vasodilators
20%
Keyphrases
Phosphodiesterase
100%
Vascular Aging
100%
ERCC1
100%
Vascular Smooth muscle Cells
50%
Genomic Instability
50%
Lung
33%
Aging
33%
Sodium Nitroprusside
33%
PDE1A
33%
Phosphodiesterase Type 5 Inhibitors
33%
PDE1
33%
Cellular Senescence
33%
Age-related Vascular Disease
33%
PDE1C
33%
Human Vascular Smooth muscle Cells
33%
Single nucleotide Polymorphism
16%
MRNA Level
16%
Genome-wide Association Study
16%
Carotid Intima-media Thickness (cIMT)
16%
Cell Population
16%
Aorta
16%
Diastolic Blood Pressure
16%
Vasoreactivity
16%
Soluble Guanylate Cyclase
16%
Soluble Guanylate Cyclase Stimulator
16%
Nitric Oxide
16%
Causal Factors
16%
Muscle Cell Culture
16%
Cyclic GMP
16%
Hydrolysis
16%
CGMP-dependent Protein Kinase
16%
Nucleotide Excision Repair
16%
CGMP Signaling
16%
Human Age
16%
DNA Repair Genes
16%
Relaxation Effect
16%
Increased Blood Pressure
16%
Cell Senescence
16%
Population Genome
16%
Vascular Hypertrophy
16%
Aortic Rings
16%
Human Genetic Studies
16%
Biochemistry, Genetics and Molecular Biology
Genome Instability
100%
Phosphodiesterase I
100%
Wild Type
66%
PDE1
66%
PDE1C
66%
PDE1A
66%
Guanylate Cyclase
66%
Nitric Oxide
33%
Single-Nucleotide Polymorphism
33%
Blood Pressure
33%
Base Excision Repair
33%
Nucleotide Excision Repair
33%
Genome-Wide Association Study
33%
Cell Culture
33%
Human Genetics
33%
Diastolic Blood Pressure
33%
Enzymatic Hydrolysis
33%
ERCC1
33%
Phosphodiesterase
33%
Gene Repair
33%
CGMP-dependent Protein Kinase
33%
Messenger RNA
33%
Pharmacology, Toxicology and Pharmaceutical Science
Vascular Disease
100%
Nitroprusside Sodium
100%
Phosphodiesterase V
100%
Phosphodiesterase I
100%
Guanylate Cyclase
100%
Nitric Oxide
50%
Elevated Blood Pressure
50%
Vasodilator Agent
50%
Phosphodiesterase
50%
Cell Culture
50%
Guanylate Cyclase Activator
50%
Cyclic GMP Dependent Protein Kinase 1
50%
Messenger RNA
50%
Medicine and Dentistry
Vascular Smooth Muscle Cell
100%
Phosphodiesterase I
100%
Genomic Instability
60%
Cellular Senescence
60%
Vascular Disease
40%
Sodium Nitroprusside
40%
Guanylate Cyclase
40%
Phosphodiesterase V
40%
Lung
40%
Nitric Oxide
20%
Cell Population
20%
Cell Culture
20%
Elevated Blood Pressure
20%
Genome Wide Association Study
20%
Vasodilator Agent
20%
Human Genetics
20%
Intima-Media Thickness
20%
Single Nucleotide Polymorphism
20%
Diastolic Blood Pressure
20%
Base Excision Repair
20%
Phosphodiesterase
20%
Gene Repair
20%
Guanylate Cyclase Activator
20%
Cyclic GMP Dependent Protein Kinase 1
20%
Messenger RNA
20%
Enzymatic Hydrolysis
20%
Immunology and Microbiology
Vascular Smooth Muscle Cell
100%
Genomic Instability
60%
Wild Type
40%
Cell Population
20%
Cell Culture
20%
Blood Pressure
20%
Diastolic Blood Pressure
20%
Single Nucleotide Polymorphism
20%
Excision Repair
20%
Genome-Wide Association Study
20%