Nitric oxide (NO) synthase but not NO, HNO or H2O2 mediates endothelium-dependent relaxation of resistance arteries from patients with cardiovascular disease

M. Matthies, K. Rosenstand, I. Nissen, S. Muitjens, L.P. Riber, J.G.R. De Mey*, M. Bloksgaard

*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

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Original languageEnglish
Pages (from-to)1049-1064
Number of pages16
JournalBritish Journal of Pharmacology
Volume179
Issue number5
Early online date16 Nov 2021
DOIs
Publication statusPublished - Mar 2022

Keywords

  • bradykinin
  • coronary artery disease
  • endothelin-1
  • oxidative stress
  • superoxide dismutase
  • BRADYKININ-INDUCED RELAXATION
  • SOLUBLE GUANYLATE-CYCLASE
  • HYDROGEN-PEROXIDE
  • HYPERPOLARIZING FACTOR
  • CONCISE GUIDE
  • CENTER-DOT
  • NITROXYL
  • VASODILATION
  • PHARMACOLOGY
  • RESPONSES

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