New insights in intestinal ischemia-reperfusion injury: implications for intestinal transplantation.

Keyphrases

• Intestinal Ischemia 100%
• Intestinal Transplantation 100%
• Intestine 66%
• Innate Immune Response 66%
• Ischemia-reperfusion Injury 66%
• Neutrophils 33%
• Pathophysiological Mechanisms 33%
• Mucosa 33%
• Mast Cells 33%
• Immune Cells 33%
• Platelets 33%
• Immune Activation 33%
• Recent Advances 33%
• Microenvironment 33%
• Therapeutic Strategies 33%
• Goblet Cells 33%
• Receptor-interacting Protein Kinase 1 (RIPK1) 33%
• Transplant Outcomes 33%
• Dendritic Cells 33%
• Pathogen-associated Molecular Patterns 33%
• Antimicrobial 33%
• Toll-like 33%
• Engraftment 33%
• Damage-associated Molecular Patterns 33%
• Injured Tissue 33%
• Danger Signals 33%
• T Cell Response 33%
• Paneth Cells 33%
• Microorganisms 33%
• Complement Proteins 33%
• Epithelial Lining Fluid 33%
• Mucus 33%
• Alloimmune Response 33%
• Intestinal Graft 33%
• Exogenous Microorganisms 33%
• Transplant Rejection 33%
• Graft Survival 33%

INIS

• injuries 100%
• ischemia 100%
• grafts 75%
• intestines 50%
• transplants 50%
• microorganisms 50%
• neutrophils 25%
• tissues 25%
• damage 25%
• receptors 25%
• design 25%
• proteins 25%
• range 25%
• mucosa 25%
• complement 25%
• mast cells 25%
• signals 25%
• host 25%
• men 25%
• shape 25%
• pathogens 25%
• linings 25%
• dendrites 25%

Immunology and Microbiology

• Reperfusion 100%
• Intestinal Transplantation 100%
• Innate Immune System 40%
• Intestine 40%
• Microorganism 40%
• Neutrophil Granulocyte 20%
• Transplant Procedure 20%
• Immunocompetent Cell 20%
• Platelet 20%
• T Cell 20%
• Transplant Rejection 20%
• Mucosa 20%
• Toll-Like Receptor 20%
• Mast Cell 20%
• Goblet Cell 20%
• Paneth Cell 20%
• Engraftment 20%
• Pathogen-Associated Molecular Pattern 20%
• Dendritic Cell 20%