NAD+ to assess health in aging humans

G.E. Janssens; R.H. Houtkooper; J. Hoeks

doi:10.18632/aging.204220

NAD+ to assess health in aging humans

G.E. Janssens
, R.H. Houtkooper
, J. Hoeks^*

^*Corresponding author for this work

Research output: Contribution to journal › Editorial › Academic › peer-review

Abstract

The molecular changes that occur in aging cells prime the organism for age-related disease. One of the changes that occur with age, extensively documented in mice, is the decline of nicotinamide adenine dinucleotide (NAD ⁺) [1]. Discovered over 100 years ago as the electron carrier for redox enzymes, and later revealed as a signaling hub, NAD ⁺ has received a surge of attention in the last decades in the aging research field [1].

Original language	English
Pages (from-to)	5962-5963
Number of pages	2
Journal	Aging
Volume	14
Issue number	15
DOIs	https://doi.org/10.18632/aging.204220
Publication status	Published - 15 Aug 2022

Keywords

NAD
healthspan
exercise
aging
frailty
EXERCISE

Access to Document

10.18632/aging.204220Licence: CC BY

Cite this

@article{51b6c0a873c64b26afe790cdb54909b3,

title = "NAD+ to assess health in aging humans",

abstract = "The molecular changes that occur in aging cells prime the organism for age-related disease. One of the changes that occur with age, extensively documented in mice, is the decline of nicotinamide adenine dinucleotide (NAD +) [1]. Discovered over 100 years ago as the electron carrier for redox enzymes, and later revealed as a signaling hub, NAD + has received a surge of attention in the last decades in the aging research field [1].",

keywords = "NAD, healthspan, exercise, aging, frailty, EXERCISE",

author = "G.E. Janssens and R.H. Houtkooper and J. Hoeks",

note = "Publisher Copyright: {\textcopyright} 2022. Janssens et al. This is an open access article distributed under the terms of the Creative Commons Attribution License (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited",

year = "2022",

month = aug,

day = "15",

doi = "10.18632/aging.204220",

language = "English",

volume = "14",

pages = "5962--5963",

journal = "Aging",

issn = "1945-4589",

publisher = "Impact Journals LLC",

number = "15",

}

TY - JOUR

T1 - NAD+ to assess health in aging humans

AU - Janssens, G.E.

AU - Houtkooper, R.H.

AU - Hoeks, J.

N1 - Publisher Copyright: © 2022. Janssens et al. This is an open access article distributed under the terms of the Creative Commons Attribution License (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited

PY - 2022/8/15

Y1 - 2022/8/15

N2 - The molecular changes that occur in aging cells prime the organism for age-related disease. One of the changes that occur with age, extensively documented in mice, is the decline of nicotinamide adenine dinucleotide (NAD +) [1]. Discovered over 100 years ago as the electron carrier for redox enzymes, and later revealed as a signaling hub, NAD + has received a surge of attention in the last decades in the aging research field [1].

AB - The molecular changes that occur in aging cells prime the organism for age-related disease. One of the changes that occur with age, extensively documented in mice, is the decline of nicotinamide adenine dinucleotide (NAD +) [1]. Discovered over 100 years ago as the electron carrier for redox enzymes, and later revealed as a signaling hub, NAD + has received a surge of attention in the last decades in the aging research field [1].

KW - NAD

KW - healthspan

KW - exercise

KW - aging

KW - frailty

KW - EXERCISE

U2 - 10.18632/aging.204220

DO - 10.18632/aging.204220

M3 - Editorial

C2 - 35939337

SN - 1945-4589

VL - 14

SP - 5962

EP - 5963

JO - Aging

JF - Aging

IS - 15

ER -

NAD+ to assess health in aging humans

Abstract

Keywords

Access to Document

Fingerprint

Cite this