Matricellular Signaling Molecule CCN1 Attenuates Experimental Autoimmune Myocarditis by Acting as a Novel Immune Cell Migration Modulator

Madlen Rother, Stefanie Krohn, Gabriela Kania, Davy Vanhoutte, Andreas Eisenreich, Xiaomin Wang, Dirk Westermann, Kostas Savvatis, Nadine Dannemann, Carsten Skurk, Denise Hilfiker-Kleiner, Toni Cathomen, Henry Fechner, Ursula Rauch, Heinz-Peter Schultheiss, Stephane Heymans, Urs Eriksson, Carmen Scheibenbogen, Wolfgang Poller

Research output: Contribution to journalArticleAcademicpeer-review

Original languageEnglish
Pages (from-to)2688-2698
JournalCirculation
Volume122
Issue number25
DOIs
Publication statusPublished - 21 Dec 2010

Keywords

  • cardiomyopathy
  • immunomodulation
  • chemotaxis
  • migration
  • inflammation

Cite this

Rother, M., Krohn, S., Kania, G., Vanhoutte, D., Eisenreich, A., Wang, X., Westermann, D., Savvatis, K., Dannemann, N., Skurk, C., Hilfiker-Kleiner, D., Cathomen, T., Fechner, H., Rauch, U., Schultheiss, H-P., Heymans, S., Eriksson, U., Scheibenbogen, C., & Poller, W. (2010). Matricellular Signaling Molecule CCN1 Attenuates Experimental Autoimmune Myocarditis by Acting as a Novel Immune Cell Migration Modulator. Circulation, 122(25), 2688-2698. https://doi.org/10.1161/CIRCULATIONAHA.110.945261