Insights in the autoimmunity of myasthenia gravis

Marc H. De Baets*

*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

2 Citations (Web of Science)


Myasthenia gravis is a prototype anti-receptor autoimmune disease. Antibodies against proteins at the neuromuscular junction cause a defect in the signal transmission from nerve terminal to the damaged postsynaptic membrane. This issue of Autoimmunity reviews the mechanisms that lead to the destruction of the neuromuscular junction and the role of the thymus in myasthenia gravis and its animal models. In addition, this issue explores recent advances in diagnosis and treatment of this disease.
Original languageEnglish
Pages (from-to)341-343
Issue number5-6
Publication statusPublished - 2010


  • Myasthenia gravis
  • autoimmunity
  • acetylcholine receptor
  • muscle-specific tyrosine kinase
  • neuromuscular

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