Increased intramyocellular lipid content but normal skeletal muscle mitochondrial oxidative capacity throughout the pathogenesis of type 2 diabetes.

H.M. Feyter*, E. Lenaers, S.M. Houten, P. Schrauwen, M.K. Hesselink, R.J. Wanders, K. Nicolay, J.J. Prompers

*Corresponding author for this work

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Biochemistry, Genetics and Molecular Biology