KRAS status is related to histological phenotype in gastric cancer: results from a large multicentre study

Lindsay C. Hewitt, Yuichi Saito, Tan Wang, Yoko Matsuda, Jan Oosting, Arnaldo N. S. Silva, Hayley L. Slaney, Veerle Melotte, Gordon Hutchins, Patrick Tan, Takaki Yoshikawa, Tomio Arai, Heike I. Grabsch*

*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

Original languageEnglish
Pages (from-to)1193-1203
Number of pages11
JournalGastric Cancer
Volume22
Issue number6
DOIs
Publication statusPublished - Nov 2019

Keywords

  • Gastric cancer
  • KRAS
  • Mutation
  • Amplification
  • Histological phenotype
  • GENE AMPLIFICATION
  • COLORECTAL-CANCER
  • MICROSATELLITE INSTABILITY
  • GENOMIC ALTERATIONS
  • MUTATIONS
  • CARCINOMA
  • BRAF
  • MICROENVIRONMENT
  • ADENOCARCINOMA
  • HETEROGENEITY

Cite this

Hewitt, Lindsay C. ; Saito, Yuichi ; Wang, Tan ; Matsuda, Yoko ; Oosting, Jan ; Silva, Arnaldo N. S. ; Slaney, Hayley L. ; Melotte, Veerle ; Hutchins, Gordon ; Tan, Patrick ; Yoshikawa, Takaki ; Arai, Tomio ; Grabsch, Heike I. / KRAS status is related to histological phenotype in gastric cancer : results from a large multicentre study. In: Gastric Cancer. 2019 ; Vol. 22, No. 6. pp. 1193-1203.
@article{89f87a93f610411dacda6eab45aef160,
title = "KRAS status is related to histological phenotype in gastric cancer: results from a large multicentre study",
keywords = "Gastric cancer, KRAS, Mutation, Amplification, Histological phenotype, GENE AMPLIFICATION, COLORECTAL-CANCER, MICROSATELLITE INSTABILITY, GENOMIC ALTERATIONS, MUTATIONS, CARCINOMA, BRAF, MICROENVIRONMENT, ADENOCARCINOMA, HETEROGENEITY",
author = "Hewitt, {Lindsay C.} and Yuichi Saito and Tan Wang and Yoko Matsuda and Jan Oosting and Silva, {Arnaldo N. S.} and Slaney, {Hayley L.} and Veerle Melotte and Gordon Hutchins and Patrick Tan and Takaki Yoshikawa and Tomio Arai and Grabsch, {Heike I.}",
year = "2019",
month = "11",
doi = "10.1007/s10120-019-00972-6",
language = "English",
volume = "22",
pages = "1193--1203",
journal = "Gastric Cancer",
issn = "1436-3291",
publisher = "Springer, Cham",
number = "6",

}

Hewitt, LC, Saito, Y, Wang, T, Matsuda, Y, Oosting, J, Silva, ANS, Slaney, HL, Melotte, V, Hutchins, G, Tan, P, Yoshikawa, T, Arai, T & Grabsch, HI 2019, 'KRAS status is related to histological phenotype in gastric cancer: results from a large multicentre study', Gastric Cancer, vol. 22, no. 6, pp. 1193-1203. https://doi.org/10.1007/s10120-019-00972-6

KRAS status is related to histological phenotype in gastric cancer : results from a large multicentre study. / Hewitt, Lindsay C.; Saito, Yuichi; Wang, Tan; Matsuda, Yoko; Oosting, Jan; Silva, Arnaldo N. S.; Slaney, Hayley L.; Melotte, Veerle; Hutchins, Gordon; Tan, Patrick; Yoshikawa, Takaki; Arai, Tomio; Grabsch, Heike I.

In: Gastric Cancer, Vol. 22, No. 6, 11.2019, p. 1193-1203.

Research output: Contribution to journalArticleAcademicpeer-review

TY - JOUR

T1 - KRAS status is related to histological phenotype in gastric cancer

T2 - results from a large multicentre study

AU - Hewitt, Lindsay C.

AU - Saito, Yuichi

AU - Wang, Tan

AU - Matsuda, Yoko

AU - Oosting, Jan

AU - Silva, Arnaldo N. S.

AU - Slaney, Hayley L.

AU - Melotte, Veerle

AU - Hutchins, Gordon

AU - Tan, Patrick

AU - Yoshikawa, Takaki

AU - Arai, Tomio

AU - Grabsch, Heike I.

PY - 2019/11

Y1 - 2019/11

KW - Gastric cancer

KW - KRAS

KW - Mutation

KW - Amplification

KW - Histological phenotype

KW - GENE AMPLIFICATION

KW - COLORECTAL-CANCER

KW - MICROSATELLITE INSTABILITY

KW - GENOMIC ALTERATIONS

KW - MUTATIONS

KW - CARCINOMA

KW - BRAF

KW - MICROENVIRONMENT

KW - ADENOCARCINOMA

KW - HETEROGENEITY

U2 - 10.1007/s10120-019-00972-6

DO - 10.1007/s10120-019-00972-6

M3 - Article

VL - 22

SP - 1193

EP - 1203

JO - Gastric Cancer

JF - Gastric Cancer

SN - 1436-3291

IS - 6

ER -