KRAS status is related to histological phenotype in gastric cancer: results from a large multicentre study

Lindsay C. Hewitt, Yuichi Saito, Tan Wang, Yoko Matsuda, Jan Oosting, Arnaldo N. S. Silva, Hayley L. Slaney, Veerle Melotte, Gordon Hutchins, Patrick Tan, Takaki Yoshikawa, Tomio Arai, Heike I. Grabsch*

*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

Original languageEnglish
Pages (from-to)1193-1203
Number of pages11
JournalGastric Cancer
Volume22
Issue number6
DOIs
Publication statusPublished - Nov 2019

Keywords

  • Gastric cancer
  • KRAS
  • Mutation
  • Amplification
  • Histological phenotype
  • GENE AMPLIFICATION
  • COLORECTAL-CANCER
  • MICROSATELLITE INSTABILITY
  • GENOMIC ALTERATIONS
  • MUTATIONS
  • CARCINOMA
  • BRAF
  • MICROENVIRONMENT
  • ADENOCARCINOMA
  • HETEROGENEITY

Cite this

Hewitt, L. C., Saito, Y., Wang, T., Matsuda, Y., Oosting, J., Silva, A. N. S., Slaney, H. L., Melotte, V., Hutchins, G., Tan, P., Yoshikawa, T., Arai, T., & Grabsch, H. I. (2019). KRAS status is related to histological phenotype in gastric cancer: results from a large multicentre study. Gastric Cancer, 22(6), 1193-1203. https://doi.org/10.1007/s10120-019-00972-6