Hippocampal ?CaMKII dopaminylation promotes synaptic-to-nuclear signaling and memory formation

Andrew F Stewart, Sasha L Fulton, Romain Durand-de Cuttoli, Robert E Thompson, Peng-Jen Chen, Elizabeth Brindley, Bulent Cetin, Lorna A Farrelly, Rita Futamura, Sarah Claypool, Ryan M Bastle, Giuseppina Di Salvo, Christopher Peralta, Henrik Molina, Erdene Baljinnyam, Samuele G Marro, Scott J Russo, Robert J DeVita, Tom W Muir*, Ian Maze*

*Corresponding author for this work

Research output: Working paper / PreprintPreprint

Abstract

Protein monoaminylation is a class of posttranslational modification (PTM) that contributes to transcription, physiology and behavior. While recent analyses have focused on histones as critical substrates of monoaminylation, the broader repertoire of monoaminylated proteins in brain remains unclear. Here, we report the development/implementation of a chemical probe for the bioorthogonal labeling, enrichment and proteomics-based detection of dopaminylated proteins in brain. We identified 1,557 dopaminylated proteins - many synaptic - including ?CaMKII, which mediates Ca -dependent cellular signaling and hippocampal-dependent memory. We found that ?CaMKII dopaminylation is largely synaptic and mediates synaptic-to-nuclear signaling, neuronal gene expression and intrinsic excitability, and contextual memory. These results indicate a critical role for synaptic dopaminylation in adaptive brain plasticity, and may suggest roles for these phenomena in pathologies associated with altered monoaminergic signaling.
Original languageEnglish
Number of pages54
DOIs
Publication statusPublished - 24 Sept 2024

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