Coronary Artery Calcium Scores After Prophylactic Premenopausal Bilateral Salpingo-Oophorectomy

Maarten J. Beekman, Lara Terra, Bernadette A.M. Heemskerk-Gerritsen, Carlijn M. van der Aalst, Jeanine E. Roeters van Lennep, Marc van Beurden, Helena C. van Doorn, Joanne A. de Hullu, Eleonora B.L. van Dorst, Constantijne H. Mom, Marian J.E. Mourits, Brigitte F.M. Slangen, Annemarieke Bartels-Rutten, Ricardo P.J. Budde, Miranda M. Snoeren, Tim Leiner, Pim A. de Jong, Rozemarijn Vliegenthart, R. Nils Planken, Casper MihlMarleen Vonder, Matthijs Oudkerk, Katja N. Gaarenstroom, Jan Willem C. Gratama, Klaartje van Engelen, Lizet E. van der Kolk, J. Margriet Collée, Marijke R. Wevers, Margreet G.E.M. Ausems, Lieke P.V. Berger, Encarna B. Gomez Garcia, Christi J. van Asperen, Maartje J. Hooning, Harry J. de Koning, Angela H.E.M. Maas, Flora E. van Leeuwen*

*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

Abstract

Background: Premenopausal risk-reducing salpingo-oophorectomy (RRSO) in women at high familial risk of ovarian cancer leads to immediate menopause. Although early natural menopause is associated with increased cardiovascular disease risk, evidence on long-term cardiovascular disease risk after early surgical menopause is scarce. Objectives: We sought to determine the long-term influence of the timing of RRSO on the development of coronary artery calcium (CAC), an established marker for cardiovascular disease risk. Methods: We conducted a cross-sectional study (N = 733) nested in a nationwide cohort of women at high familial risk of ovarian cancer. In women aged 60-70 years (n = 328), we compared CAC scores between women with a premenopausal RRSO (age ≤45 years) and women with a postmenopausal RRSO (age ≥54 years), using multivariable Poisson analyses. Within the premenopausal RRSO group (n = 498), we also examined the effect of age at RRSO. In addition, we compared the premenopausal RRSO group with an external reference cohort (n = 5,226). Results: Multivariable analyses showed that the prevalence rates of any CAC (CAC >0), at least moderate CAC (CAC >100), and severe CAC (CAC >400) were comparable between the premenopausal and postmenopausal RRSO groups (relative risk [RR]: 0.93; 95% CI: 0.75-1.15 for any CAC; RR: 0.71; 95% CI: 0.43-1.17 for at least moderate CAC; RR: 0.81; 95% CI: 0.30-2.13 for severe CAC). There was no difference in CAC between the premenopausal RRSO group and a similar aged reference cohort. Timing of premenopausal RRSO (early premenopausal RRSO [<41 years] vs late premenopausal RRSO [41-45 years]) did not affect the outcomes. Conclusions: Our results do not show a long-term adverse effect of surgical menopause on the development of CAC.

Original languageEnglish
Pages (from-to)922-931
Number of pages10
JournalJACC: CardioOncology
Volume6
Issue number6
Early online date1 Dec 2024
DOIs
Publication statusPublished - Dec 2024

Keywords

  • BRCA
  • CAC
  • cardiovascular disease
  • ovarian cancer
  • RRSO
  • surgical menopause

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