INIS
apoptosis
17%
biological markers
34%
capacity
18%
cardiovascular diseases
47%
comparative evaluations
20%
control
36%
diseases
49%
failures
16%
fibrosis
18%
genes
73%
genetics
17%
heart failure
100%
humans
44%
hypertrophy
65%
in vivo
17%
levels
31%
mice
75%
molecules
16%
muscles
16%
myocardial infarction
19%
myocardium
21%
patients
30%
phenotype
17%
proteins
24%
receptors
21%
regulations
29%
reviews
22%
rna
79%
therapy
25%
tissues
17%
Keyphrases
Cardiac Development
12%
Cardiac Disease
10%
Cardiac Dysfunction
15%
Cardiac Function
16%
Cardiac Hypertrophy
30%
Cardiac Remodeling
19%
Cardiomyocytes
36%
Cardiovascular Disease
41%
Circulating miRs
13%
Epigenetics
13%
European Society of Cardiology
17%
Extracellular Vesicles
11%
Fibrosis
11%
Heart Failure
81%
Hypertrophy
14%
Hypoxia
11%
Induced Pluripotent Stem Cells (iPSCs)
12%
Long Non-coding RNA (LncRNA)
10%
MicroRNA
89%
Myocardial Function
23%
Myocardial Infarction
14%
Myocardium
16%
Non-coding RNA (ncRNA)
50%
On-a-chip
13%
Pathophysiology
11%
Position Paper
21%
Preeclampsia
13%
Pressure Overload
30%
Therapeutic Target
11%
Transcription Factor
13%
Medicine and Dentistry
Aortic Constriction
9%
Apoptosis Inducing Factor
8%
Biological Marker
10%
Calcineurin
11%
Cardiac Muscle Cell
21%
Cardiology
17%
Cardiovascular Disease
13%
Cardiovascular System
11%
Cell Death
7%
Congestive Heart Failure
66%
Disease
7%
Endothelial Cell
10%
Fibrosis
8%
Gene Expression
12%
Heart Function
38%
Heart Ventricle Remodeling
21%
Hypoxia
12%
Left Ventricle
8%
Long Untranslated RNA
9%
microRNA
46%
microRNA 200c
8%
Myocardium
7%
Pathophysiology
8%
Peroxisome Proliferator Activated Receptor Alpha
8%
Pre-Eclampsia
8%
Right Ventricle
8%
Skeletal Muscle
8%
Transcription Factors
10%
Untranslated RNA
10%
Ventricular Hypertrophy
20%
